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nf κb inhibitor imd 0354  (MedChemExpress)


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    MedChemExpress nf κb inhibitor imd 0354
    Nf κb Inhibitor Imd 0354, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 26 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 94 stars, based on 26 article reviews
    nf κb inhibitor imd 0354 - by Bioz Stars, 2026-02
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    Fig. 6. The PM2.5-induced increase in α-SMA was dependent on NF-κB, but not AhR signaling. A and B) Fibroblasts were exposed to repeated treatment of PM2.5 from Beijing (A) or NIST (B) in the presence or absence of 10 µM of the the AhR inhibitor CH223191. Levels of α-SMA and collagen 1a1 were analyzed by immunoblot. Representative immunoblots from two independent experiments from both Beijing and NIST PM2.5 are shown. C) and D) Fibroblasts given repeated treatment of PM2.5 from Beijing (C) or NIST (D) in the presence or absence of 100 nM of the NF-κB inhibitor <t>IMD0354</t> were analyzed for α-SMA or collagen 1a1 expression by immunoblot. Shown are representative blots from two (C) and three (D) independent experiments with densitometric analysis relative to α-tubulin shown below. * P < 0.05, * * P < 0.01.
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    Fig. 6. The PM2.5-induced increase in α-SMA was dependent on NF-κB, but not AhR signaling. A and B) Fibroblasts were exposed to repeated treatment of PM2.5 from Beijing (A) or NIST (B) in the presence or absence of 10 µM of the the AhR inhibitor CH223191. Levels of α-SMA and collagen 1a1 were analyzed by immunoblot. Representative immunoblots from two independent experiments from both Beijing and NIST PM2.5 are shown. C) and D) Fibroblasts given repeated treatment of PM2.5 from Beijing (C) or NIST (D) in the presence or absence of 100 nM of the NF-κB inhibitor <t>IMD0354</t> were analyzed for α-SMA or collagen 1a1 expression by immunoblot. Shown are representative blots from two (C) and three (D) independent experiments with densitometric analysis relative to α-tubulin shown below. * P < 0.05, * * P < 0.01.
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    Fig. 6. The PM2.5-induced increase in α-SMA was dependent on NF-κB, but not AhR signaling. A and B) Fibroblasts were exposed to repeated treatment of PM2.5 from Beijing (A) or NIST (B) in the presence or absence of 10 µM of the the AhR inhibitor CH223191. Levels of α-SMA and collagen 1a1 were analyzed by immunoblot. Representative immunoblots from two independent experiments from both Beijing and NIST PM2.5 are shown. C) and D) Fibroblasts given repeated treatment of PM2.5 from Beijing (C) or NIST (D) in the presence or absence of 100 nM of the NF-κB inhibitor <t>IMD0354</t> were analyzed for α-SMA or collagen 1a1 expression by immunoblot. Shown are representative blots from two (C) and three (D) independent experiments with densitometric analysis relative to α-tubulin shown below. * P < 0.05, * * P < 0.01.
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    Fig. 6. The PM2.5-induced increase in α-SMA was dependent on NF-κB, but not AhR signaling. A and B) Fibroblasts were exposed to repeated treatment of PM2.5 from Beijing (A) or NIST (B) in the presence or absence of 10 µM of the the AhR inhibitor CH223191. Levels of α-SMA and collagen 1a1 were analyzed by immunoblot. Representative immunoblots from two independent experiments from both Beijing and NIST PM2.5 are shown. C) and D) Fibroblasts given repeated treatment of PM2.5 from Beijing (C) or NIST (D) in the presence or absence of 100 nM of the NF-κB inhibitor IMD0354 were analyzed for α-SMA or collagen 1a1 expression by immunoblot. Shown are representative blots from two (C) and three (D) independent experiments with densitometric analysis relative to α-tubulin shown below. * P < 0.05, * * P < 0.01.

    Journal: Environmental toxicology and pharmacology

    Article Title: Promotion of myofibroblast differentiation through repeated treatment of fibroblasts to low concentrations of PM 2.5 .

    doi: 10.1016/j.etap.2023.104329

    Figure Lengend Snippet: Fig. 6. The PM2.5-induced increase in α-SMA was dependent on NF-κB, but not AhR signaling. A and B) Fibroblasts were exposed to repeated treatment of PM2.5 from Beijing (A) or NIST (B) in the presence or absence of 10 µM of the the AhR inhibitor CH223191. Levels of α-SMA and collagen 1a1 were analyzed by immunoblot. Representative immunoblots from two independent experiments from both Beijing and NIST PM2.5 are shown. C) and D) Fibroblasts given repeated treatment of PM2.5 from Beijing (C) or NIST (D) in the presence or absence of 100 nM of the NF-κB inhibitor IMD0354 were analyzed for α-SMA or collagen 1a1 expression by immunoblot. Shown are representative blots from two (C) and three (D) independent experiments with densitometric analysis relative to α-tubulin shown below. * P < 0.05, * * P < 0.01.

    Article Snippet: In some experiments, the cells were pre-incubated with the AhR antagonist CH223191 (10 μM, Tocris Bioscience, Ellisville, MO) or the NF-κB inhibitor IMD0354 (10 nM, Tocris Bioscience) for 30 min prior to addition of PM2.5.

    Techniques: Western Blot, Expressing